The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 85
Experimentally , amyloidosis has been produced by feeding large amounts of protein , especially cheese or casein , and it has been claimed that the disease is rare in countries with little milk or cheese consumption , whereas it is a ...
Experimentally , amyloidosis has been produced by feeding large amounts of protein , especially cheese or casein , and it has been claimed that the disease is rare in countries with little milk or cheese consumption , whereas it is a ...
Page 86
Galactosemia , another rare , inborn disturbance of the carbohydrate metabolism , is assumedly caused by a defect of a hepatic enzyme required for the transformation of galactose - 1 - phosphate to glucose - 1 - phosphate .
Galactosemia , another rare , inborn disturbance of the carbohydrate metabolism , is assumedly caused by a defect of a hepatic enzyme required for the transformation of galactose - 1 - phosphate to glucose - 1 - phosphate .
Page 141
It is rare that more than a single nodule can be detected . The structure is usually similar to that of the normal pancreas but may become cystic , and instances where the mass was primarily or entirely made up of islet cells have been ...
It is rare that more than a single nodule can be detected . The structure is usually similar to that of the normal pancreas but may become cystic , and instances where the mass was primarily or entirely made up of islet cells have been ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall