The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 143
... region and muscular resistance may be found . This stage may clear com- pletely but may recur with more severe symptoms and damage to the pancreas . When the process progresses - usually rapidly to acute hemorrhagic pancre- atitis , the ...
... region and muscular resistance may be found . This stage may clear com- pletely but may recur with more severe symptoms and damage to the pancreas . When the process progresses - usually rapidly to acute hemorrhagic pancre- atitis , the ...
Page 147
... region of the other with organs . pressure on development and growth from benign cystadenoma may be relatively slow , with case histories of known duration of sev- eral years not being very uncommon . Several have been reported to ...
... region of the other with organs . pressure on development and growth from benign cystadenoma may be relatively slow , with case histories of known duration of sev- eral years not being very uncommon . Several have been reported to ...
Page 149
... regional nodes along the common duct , the peri- aortic nodes , the liver and , via the gastric nodes , the mediastinum and neck . Direct spread also takes place to the stomach , colon , spleen and kidney , and ero- sion through the ...
... regional nodes along the common duct , the peri- aortic nodes , the liver and , via the gastric nodes , the mediastinum and neck . Direct spread also takes place to the stomach , colon , spleen and kidney , and ero- sion through the ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall