The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 38
... serum or plasma constituents with the so - called hepatic tests will disclose an isolated hepatic deficiency restricted to the protein metabolism . Abnormalities of serum proteins almost always reflect a far more general disturbance of ...
... serum or plasma constituents with the so - called hepatic tests will disclose an isolated hepatic deficiency restricted to the protein metabolism . Abnormalities of serum proteins almost always reflect a far more general disturbance of ...
Page 42
... serum , where their solubility is influenced by phospholipids , serum bile acids and proteins , all three produced by the liver . Although the liver is the main source of serum cholesterol , its level is , of course , influenced by a ...
... serum , where their solubility is influenced by phospholipids , serum bile acids and proteins , all three produced by the liver . Although the liver is the main source of serum cholesterol , its level is , of course , influenced by a ...
Page 58
... serum amylase , Somogyi's has found widest use . Plasma or serum is added to a starch solution , and the glucose content over and above the reducing power of the serum before it was incubated with starch is determined . The amylase ...
... serum amylase , Somogyi's has found widest use . Plasma or serum is added to a starch solution , and the glucose content over and above the reducing power of the serum before it was incubated with starch is determined . The amylase ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall