The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 90
... severe , central necrosis develops with or without disappearance of the liver cells , which are usually replaced by segmented leukocytes . Diverse factors can be respon- sible for such changes : ( 1 ) general cir- culatory failure ...
... severe , central necrosis develops with or without disappearance of the liver cells , which are usually replaced by segmented leukocytes . Diverse factors can be respon- sible for such changes : ( 1 ) general cir- culatory failure ...
Page 95
... severe hepatic insufficiency , dominated by central nerv- ous system manifestations and severe hemorrhages . Sometimes only the ter- minal period is associated with severe jaundice . The laboratory findings depend greatly upon the stage ...
... severe hepatic insufficiency , dominated by central nerv- ous system manifestations and severe hemorrhages . Sometimes only the ter- minal period is associated with severe jaundice . The laboratory findings depend greatly upon the stage ...
Page 143
... severe symptoms and damage to the pancreas . When the process progresses - usually rapidly to acute hemorrhagic pancre- atitis , the patients become severely ill , with excruciating pain radiating to the back , and spasm or even ...
... severe symptoms and damage to the pancreas . When the process progresses - usually rapidly to acute hemorrhagic pancre- atitis , the patients become severely ill , with excruciating pain radiating to the back , and spasm or even ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall