The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 90
If shock is more severe , central necrosis develops with or without disappearance
of the liver cells , which are usually replaced by segmented leukocytes . Diverse
factors can be responsible for such changes : ( 1 ) general circulatory failure ...
If shock is more severe , central necrosis develops with or without disappearance
of the liver cells , which are usually replaced by segmented leukocytes . Diverse
factors can be responsible for such changes : ( 1 ) general circulatory failure ...
Page 95
A short preicteric period is usually followed by a long icteric phase and a terminal
period of acute severe hepatic insufficiency , dominated by central nervous
system manifestations and severe hemorrhages . Sometimes only the terminal ...
A short preicteric period is usually followed by a long icteric phase and a terminal
period of acute severe hepatic insufficiency , dominated by central nervous
system manifestations and severe hemorrhages . Sometimes only the terminal ...
Page 143
This stage may clear completely but may recur with more severe symptoms and
damage to the pancreas . When the process progresses – usually rapidly – to
acute hemorrhagic pancreatitis , the patients become severely ill , with
excruciating ...
This stage may clear completely but may recur with more severe symptoms and
damage to the pancreas . When the process progresses – usually rapidly – to
acute hemorrhagic pancreatitis , the patients become severely ill , with
excruciating ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall