The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 90
If shock is more severe , central necrosis develops with or without disappearance of the liver cells , which are usually replaced by segmented leukocytes . Diverse factors can be responsible for such changes : ( 1 ) general circulatory ...
If shock is more severe , central necrosis develops with or without disappearance of the liver cells , which are usually replaced by segmented leukocytes . Diverse factors can be responsible for such changes : ( 1 ) general circulatory ...
Page 95
A short preicteric period is usually followed by a long icteric phase and a terminal period of acute severe hepatic insufficiency , dominated by central nervous system manifestations and severe hemorrhages . Sometimes only the terminal ...
A short preicteric period is usually followed by a long icteric phase and a terminal period of acute severe hepatic insufficiency , dominated by central nervous system manifestations and severe hemorrhages . Sometimes only the terminal ...
Page 143
This stage may clear completely but may recur with more severe symptoms and damage to the pancreas . When the process progresses – usually rapidly – to acute hemorrhagic pancreatitis , the patients become severely ill ...
This stage may clear completely but may recur with more severe symptoms and damage to the pancreas . When the process progresses – usually rapidly – to acute hemorrhagic pancreatitis , the patients become severely ill ...
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Contents
NORMAL ANATOMY OF THE LIVER | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
18 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations Amer appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice latter leads lesions less liver cells lobe lobular lobule manifestations necrosis Netter nodes nodules normal observed obstruction occurs organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space stage stones superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall