The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 48
... skin is of a reddish hue superimposed on the pale appearance of the skin of an anemic patient . The prompt reacting bili- rubin is not elevated , and no bilirubin appears in the urine , because the indirect bilirubin can be taken up by ...
... skin is of a reddish hue superimposed on the pale appearance of the skin of an anemic patient . The prompt reacting bili- rubin is not elevated , and no bilirubin appears in the urine , because the indirect bilirubin can be taken up by ...
Page 89
... skin , liver or gastric mucosa by biopsies , of which the liver biopsy has recently received preference . The presence of the classical triad brown , atrophic skin , diabetes and the sometimes enlarged and , in later stages , firm and ...
... skin , liver or gastric mucosa by biopsies , of which the liver biopsy has recently received preference . The presence of the classical triad brown , atrophic skin , diabetes and the sometimes enlarged and , in later stages , firm and ...
Page 103
... skin and produces suppura- tion . The typical initial localizations of the abscesses are the jaws , the lung and the intestine , especially cecum and appendix . From the primary localization the suppuration spreads into the vicin- ity ...
... skin and produces suppura- tion . The typical initial localizations of the abscesses are the jaws , the lung and the intestine , especially cecum and appendix . From the primary localization the suppuration spreads into the vicin- ity ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall