The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 28
... spleen and kidney and becomes fixed in the lienorenal ligament . After its rota- tion the transverse mesocolon becomes fused with the omental bursa and so comes to lie along the inferior border and across the head of the pancreas . The ...
... spleen and kidney and becomes fixed in the lienorenal ligament . After its rota- tion the transverse mesocolon becomes fused with the omental bursa and so comes to lie along the inferior border and across the head of the pancreas . The ...
Page 69
... spleen , or by determining the wedge pressure in the hepatic veins by transcardiac catheteriza- tion ) , a variety of collaterals between portal vein and caval systems come into existence ( see also pages 18 , 72 and 73 ) . Some of them ...
... spleen , or by determining the wedge pressure in the hepatic veins by transcardiac catheteriza- tion ) , a variety of collaterals between portal vein and caval systems come into existence ( see also pages 18 , 72 and 73 ) . Some of them ...
Page 72
... SPLEEN MODERATELY ENLARGED ESOPHAGEAL VARICES SPLEEN MARKEDLY ENLARGED INTRAHEPATIC CAUSES The portal venous pressure rises above the norm of approximately 20 cm . of water because of ( 1 ) block in the intra- hepatic portal vein tree ...
... SPLEEN MODERATELY ENLARGED ESOPHAGEAL VARICES SPLEEN MARKEDLY ENLARGED INTRAHEPATIC CAUSES The portal venous pressure rises above the norm of approximately 20 cm . of water because of ( 1 ) block in the intra- hepatic portal vein tree ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall