The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. Oppenheimer |
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Page 124
SURFACE ASPECT HYPERCHOLESTEREMIA SECTION DIABETES DIABETES
PREGNANCY CHOLESTEROL STONES RADIOTRANSPARENT (
CHOLECYSTOGRAM ) METABOLIC FACTORS OBESITY CHOLELITHIASIS
Stone ...
SURFACE ASPECT HYPERCHOLESTEREMIA SECTION DIABETES DIABETES
PREGNANCY CHOLESTEROL STONES RADIOTRANSPARENT (
CHOLECYSTOGRAM ) METABOLIC FACTORS OBESITY CHOLELITHIASIS
Stone ...
Page 127
Moreover , calcium salts are released into the lumen and produce either a fine
precipitation ( milk of calcium bile ) or calcium stones . The calcium salts may also
form a shell around the stone impacted at the orifice of the cystic duct , thereby ...
Moreover , calcium salts are released into the lumen and produce either a fine
precipitation ( milk of calcium bile ) or calcium stones . The calcium salts may also
form a shell around the stone impacted at the orifice of the cystic duct , thereby ...
Page 132
Large - bowel obstruction by stones is less likely than severe biliary tract
infections with chills and fever , weight loss and jaundice . The latter may
ameliorate coincidentally with the appearance of diarrhea . Barium enemas
usually reveal the ...
Large - bowel obstruction by stones is less likely than severe biliary tract
infections with chills and fever , weight loss and jaundice . The latter may
ameliorate coincidentally with the appearance of diarrhea . Barium enemas
usually reveal the ...
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Contents
Section XV | 1 |
Cholelithiasis I Stone Formation | 2 |
Prenatal and Postnatal Circulation | 3 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage depending develop diagnosis dilated disease duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice latter lead lesions less liver cells lobe lobular manifestations mesenteric necrosis nodes nodules normal obstruction occur organ origin pain pancreatic passes patients picture plates portal vein portion posterior present primary produce protein rare result seen serum severe sinusoids sometimes space sphincter splenic stage stones structures superior surface surrounding tests tion tissue tract triads tumor usually vary vessels wall