The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 48
... STOOL DARK ₤ Nitter M.D. OCIBA BOWEL the greater intestinal concentration , so that the liver , probably somewhat damaged by the anemia , can no longer handle the large quantities of returned urobilinogen , which pass into the circu ...
... STOOL DARK ₤ Nitter M.D. OCIBA BOWEL the greater intestinal concentration , so that the liver , probably somewhat damaged by the anemia , can no longer handle the large quantities of returned urobilinogen , which pass into the circu ...
Page 49
... STOOL LIGHT BLOOD STREAM KIDNEY URINE BILIRUBIN ++++ UROBILINOGEN ? -JAUNDICE MEDIUM TO DEEP GREENISH - YELLOW STOOL LIGHT ( Continued from page 48 ) hepatic cholestasis , which , however , develops more often without demonstrable ...
... STOOL LIGHT BLOOD STREAM KIDNEY URINE BILIRUBIN ++++ UROBILINOGEN ? -JAUNDICE MEDIUM TO DEEP GREENISH - YELLOW STOOL LIGHT ( Continued from page 48 ) hepatic cholestasis , which , however , develops more often without demonstrable ...
Page 58
... STOOL FAT 10 100 200 gm . INTAKE / DAY 20 N 10 10 20 gm . INTAKE / DAY 100 200 20 10- 10 20 MODERATE DECREASE IN VOLUME , [ HCO , AND AMYLASE MARKED DECREASE IN VOLUME , [ HCO , AND AMYLASE 20 RANGE 100 200 20- 10- RANGE 10 20 40 20 100 ...
... STOOL FAT 10 100 200 gm . INTAKE / DAY 20 N 10 10 20 gm . INTAKE / DAY 100 200 20 10- 10 20 MODERATE DECREASE IN VOLUME , [ HCO , AND AMYLASE MARKED DECREASE IN VOLUME , [ HCO , AND AMYLASE 20 RANGE 100 200 20- 10- RANGE 10 20 40 20 100 ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall