The Ciba Collection of Medical Illustrations: A compilation of paintings on the normal and pathologic anatomy of the digestive system. pt. 1. Upper digestive tract. pt. 2. Lower digestive tract. pt. 3. Liver, biliary tract and pancreas. Edited by E. OppenheimerCiba Pharmaceutical Products, 1957 - Anatomy, Pathological |
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Page 10
... triads . In these the branches of the portal vein terminate , while the smallest arterioles and ductules , and probably also lymphatics ( see page 20 ) , still fre- quently covered with a thin sheet of con- nective tissue , extend into ...
... triads . In these the branches of the portal vein terminate , while the smallest arterioles and ductules , and probably also lymphatics ( see page 20 ) , still fre- quently covered with a thin sheet of con- nective tissue , extend into ...
Page 83
... triads takes place , with some proliferation of perilobular cholangioles and periportal ducts . An inflammatory reaction ensues , without bacterial infec- tion , probably as a result of a reaction to the bile stasis . As a rule , in ...
... triads takes place , with some proliferation of perilobular cholangioles and periportal ducts . An inflammatory reaction ensues , without bacterial infec- tion , probably as a result of a reaction to the bile stasis . As a rule , in ...
Page 92
... triads , espe- cially around the lymphatics . More often than by any other mode of transmission , bacteria reach the portal triads by way of the lymphatics and arterial blood , when the primary infection spreads from else- where in the ...
... triads , espe- cially around the lymphatics . More often than by any other mode of transmission , bacteria reach the portal triads by way of the lymphatics and arterial blood , when the primary infection spreads from else- where in the ...
Contents
SECTION XV | 1 |
Peritoneal Relations of Pancreas | 63 |
Hypoxic Conditions | 90 |
Copyright | |
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior primary hepatic protein result right hepatic SECTION XVII-PLATE septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall