Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 42
... disease among relatives of patients with ulcerative colitis . Scand J Gastroenterol 1987 ; 22 : 214–218 . 36. Korelitz Bl . From Crohn to Crohn's disease : 1979. An epidemiologic study in New York City . Mt Sinai J Med ( NY ) 1979 ; 46 ...
... disease among relatives of patients with ulcerative colitis . Scand J Gastroenterol 1987 ; 22 : 214–218 . 36. Korelitz Bl . From Crohn to Crohn's disease : 1979. An epidemiologic study in New York City . Mt Sinai J Med ( NY ) 1979 ; 46 ...
Page 109
... Crohn's ileitis ( n = 4 ) IgA 79 % ( x2.8 ) IgM 13 % ( x3.3 ) 134 IC / unit IgA 53 % ( x1.7 ) ( x3 ) IgG 8 % ( x4.1 ) ... colitis ( n = 11 ) 407 IC / unit ( x2.8 ) Severe Crohn's IgA 57 % ( x9 ) IgM 15 % ( x12 ) ileitis remote from fissure ( n ...
... Crohn's ileitis ( n = 4 ) IgA 79 % ( x2.8 ) IgM 13 % ( x3.3 ) 134 IC / unit IgA 53 % ( x1.7 ) ( x3 ) IgG 8 % ( x4.1 ) ... colitis ( n = 11 ) 407 IC / unit ( x2.8 ) Severe Crohn's IgA 57 % ( x9 ) IgM 15 % ( x12 ) ileitis remote from fissure ( n ...
Page 562
... colitis Steroid sparing Refractory disease Crohn's disease Steroid sparing Fistulas Refractory disease Perineal disease Maintenance of remission Toxic effects Rash and fever Bone marrow depression Pancreatitis Hepatitis ? Risk of ...
... colitis Steroid sparing Refractory disease Crohn's disease Steroid sparing Fistulas Refractory disease Perineal disease Maintenance of remission Toxic effects Rash and fever Bone marrow depression Pancreatitis Hepatitis ? Risk of ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro