Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 161
... Gastroenterology 1989 ; 96 : 313 . Abstract . 159. Pullman W , Hapel AJ , Doe WF . Colony stimulating factor production by intestinal lamina propria cells is increased in inflammatory bowel disease . Gastroenterology 1988 ; 94 : 361 ...
... Gastroenterology 1989 ; 96 : 313 . Abstract . 159. Pullman W , Hapel AJ , Doe WF . Colony stimulating factor production by intestinal lamina propria cells is increased in inflammatory bowel disease . Gastroenterology 1988 ; 94 : 361 ...
Page 218
... Gastroenterology 1989 ; 96 : 1290-1300 . 31. Rachmilewitz D , Simon PL , Schwartz LW , Griswold DE , Fondacaro JD , Wasserman MA . Inflammatory mediators of experimental colitis in rats . Gastroenterology 1989 ; 97 : 326-337 . 32 ...
... Gastroenterology 1989 ; 96 : 1290-1300 . 31. Rachmilewitz D , Simon PL , Schwartz LW , Griswold DE , Fondacaro JD , Wasserman MA . Inflammatory mediators of experimental colitis in rats . Gastroenterology 1989 ; 97 : 326-337 . 32 ...
Page 352
... Gastroenterology 1987 ; 93 : 44–52 . 38. Robert A , Asano T. Resistance of germfree rats to indomethacin - induced intestinal lesions . Prostaglandins 1977 ; 14 : 333–341 . 39. Fang W , Broughton A , Jacobson ED . Indomethacin - induced ...
... Gastroenterology 1987 ; 93 : 44–52 . 38. Robert A , Asano T. Resistance of germfree rats to indomethacin - induced intestinal lesions . Prostaglandins 1977 ; 14 : 333–341 . 39. Fang W , Broughton A , Jacobson ED . Indomethacin - induced ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro