Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 25
... IBD in persons who have a genetic predisposition to these diseases . Ankylosing Spondylitis 90-94 There is no doubt that the proportion of UC and CD patients who have ankylosing spondylitis is much higher than one might expect to find ...
... IBD in persons who have a genetic predisposition to these diseases . Ankylosing Spondylitis 90-94 There is no doubt that the proportion of UC and CD patients who have ankylosing spondylitis is much higher than one might expect to find ...
Page 26
... IBD patients in whom clinical bowel disease is not apparent but in whom sub- clinical bowel disease leads to spondylitis . Thus , IBD is associated with factors that increase the risk for ankylosing spondylitis in persons genetically ...
... IBD patients in whom clinical bowel disease is not apparent but in whom sub- clinical bowel disease leads to spondylitis . Thus , IBD is associated with factors that increase the risk for ankylosing spondylitis in persons genetically ...
Page 307
... IBD may lead to confusion.45 An individual with presumed IBS may ultimately be found to have IBD as the cause of their bowel symptoms . Likewise , it has been shown that one third of patients with quiescent IBD experience IBS - like ...
... IBD may lead to confusion.45 An individual with presumed IBS may ultimately be found to have IBD as the cause of their bowel symptoms . Likewise , it has been shown that one third of patients with quiescent IBD experience IBS - like ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro