Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 12
... J. Ulcerative colitis in the Jewish population of southern Israel 1961-1985 ... Scand Suppl 1979 ; 490 : 1-84 . 22. Kyle J , Stark G. Fall in the incidence of Crohn's disease . Gut 1980 ; 21 : 340–343 . 23. Bergman L , Krause U. The incidence ...
... J. Ulcerative colitis in the Jewish population of southern Israel 1961-1985 ... Scand Suppl 1979 ; 490 : 1-84 . 22. Kyle J , Stark G. Fall in the incidence of Crohn's disease . Gut 1980 ; 21 : 340–343 . 23. Bergman L , Krause U. The incidence ...
Page 14
... Scand J Gastroenterol 1987 ; 22 : 1009-1024 . 69. Ekbom A , Adami HO , Helmick CG , Jonzon A , Zack MM . Perinatal risk factors for inflammatory bowel disease : A case - control study . Am J Epidemiol 1990 ; 132 : 1111– 1119 . 70 ...
... Scand J Gastroenterol 1987 ; 22 : 1009-1024 . 69. Ekbom A , Adami HO , Helmick CG , Jonzon A , Zack MM . Perinatal risk factors for inflammatory bowel disease : A case - control study . Am J Epidemiol 1990 ; 132 : 1111– 1119 . 70 ...
Page 72
... J Dig Dis 1973 ; 18 : 641 . 87. Deodhar SD , Michener WM , Farmer RG . A study of the immunologic aspects of chronic ... Scand J Gastroenterol 1983 ; 18 : 305 . 94. Swarbrick ET et al . Chlamydia , cytomegalovirus and Yersinia in inflammatory ...
... J Dig Dis 1973 ; 18 : 641 . 87. Deodhar SD , Michener WM , Farmer RG . A study of the immunologic aspects of chronic ... Scand J Gastroenterol 1983 ; 18 : 305 . 94. Swarbrick ET et al . Chlamydia , cytomegalovirus and Yersinia in inflammatory ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro