Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 50
... Shanahan F. Neutrophil autoan- tibodies in ulcerative colitis : Comparison with other colitides / diarrheal diseases . Gastroenterology 1990 ; 98 : A167 . 220. Duerr RH , Targan SR , Landers CJ , Sutherland LR , Shanahan F. Neutrophil ...
... Shanahan F. Neutrophil autoan- tibodies in ulcerative colitis : Comparison with other colitides / diarrheal diseases . Gastroenterology 1990 ; 98 : A167 . 220. Duerr RH , Targan SR , Landers CJ , Sutherland LR , Shanahan F. Neutrophil ...
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Richard P. MacDermott, William F. Stenson. 5. MacDermott RP , Stenson WF . Inflammatory bowel disease . In : Targan S , Shanahan F , eds . Immunology and Immunopathology of the Liver and Gastrointestinal Tract . New York / Tokyo : Igaku ...
Richard P. MacDermott, William F. Stenson. 5. MacDermott RP , Stenson WF . Inflammatory bowel disease . In : Targan S , Shanahan F , eds . Immunology and Immunopathology of the Liver and Gastrointestinal Tract . New York / Tokyo : Igaku ...
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... Shanahan F , Landers C , Ganz T , Targan S. A subset of antineutrophil an- ticytoplasmic antibodies is associated with inflammatory bowel disease . J Allergy Clin Immunol 1990 ; 86 : 202-210 . 71. Duerr RH , Targan SR , Landers CJ , et ...
... Shanahan F , Landers C , Ganz T , Targan S. A subset of antineutrophil an- ticytoplasmic antibodies is associated with inflammatory bowel disease . J Allergy Clin Immunol 1990 ; 86 : 202-210 . 71. Duerr RH , Targan SR , Landers CJ , et ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro