Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 62
... T - cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access ... T cell - dependent and T cell - independent pathways 62 L. MAYER.
... T - cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access ... T cell - dependent and T cell - independent pathways 62 L. MAYER.
Page 81
... T Cells In contrast to other mechanisms of lymphocyte - mediated cytotoxicity , little is known of cytotoxic T - cell function in the human mucosa . Mitogen - induced cel- lular cytotoxicity has been used as an indirect measure of T - cell ...
... T Cells In contrast to other mechanisms of lymphocyte - mediated cytotoxicity , little is known of cytotoxic T - cell function in the human mucosa . Mitogen - induced cel- lular cytotoxicity has been used as an indirect measure of T - cell ...
Page 84
... cell that might facilitate or drive the activation of mucosal T cells.75 Intestinal ep- ithelial cells have been shown to be antigen - presenting cells . Unlike conven- tional antigen - presenting cells , the intestinal epithelial cell ...
... cell that might facilitate or drive the activation of mucosal T cells.75 Intestinal ep- ithelial cells have been shown to be antigen - presenting cells . Unlike conven- tional antigen - presenting cells , the intestinal epithelial cell ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro