Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 31
... abnormalities were confined to families of probands with similar abnormalities , suggesting that ( 1 ) the abnormalities are not simply secondary to the CD , ( 2 ) the abnormalities are familial , and ( 3 ) as these occurred in only ...
... abnormalities were confined to families of probands with similar abnormalities , suggesting that ( 1 ) the abnormalities are not simply secondary to the CD , ( 2 ) the abnormalities are familial , and ( 3 ) as these occurred in only ...
Page 33
... abnormalities in unaffected family members similar to those found in the probands can distinguish between an inherited predisposition and a secondary abnormality resulting from the disease process . Finding such abnormalities in ...
... abnormalities in unaffected family members similar to those found in the probands can distinguish between an inherited predisposition and a secondary abnormality resulting from the disease process . Finding such abnormalities in ...
Page 400
... abnormalities including acute pancreatitis and pancreatitic insuffi- ciency have been observed in a small number of inflammatory bowel disease patients.71 Some of these inflammatory bowel disease patients have risk factors for ...
... abnormalities including acute pancreatitis and pancreatitic insuffi- ciency have been observed in a small number of inflammatory bowel disease patients.71 Some of these inflammatory bowel disease patients have risk factors for ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro