Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 295
... acid metab- olism in human neutrophils and free radical scavenging . Adu Prostaglandin Throm- boxane Leukotriene Res 1987 ; 17 : 918-922 . 110. Hawkey CH , Boughton - Smith NK , Whittle BJR . Modulation of human colonic arach- idonic acid ...
... acid metab- olism in human neutrophils and free radical scavenging . Adu Prostaglandin Throm- boxane Leukotriene Res 1987 ; 17 : 918-922 . 110. Hawkey CH , Boughton - Smith NK , Whittle BJR . Modulation of human colonic arach- idonic acid ...
Page 593
... acid . Br Med J 1982 ; 295 : 1012 . 68. Dew MJ , Harries AD , Evans N , Evans BK , Rhodes J. Maintenance of remission in ulcerative colitis with 5 - aminosalicylic acid in high doses by mouth . Br Med J 1984 ; 287 : 23-24 . 69. Riley SA ...
... acid . Br Med J 1982 ; 295 : 1012 . 68. Dew MJ , Harries AD , Evans N , Evans BK , Rhodes J. Maintenance of remission in ulcerative colitis with 5 - aminosalicylic acid in high doses by mouth . Br Med J 1984 ; 287 : 23-24 . 69. Riley SA ...
Page 595
... acid enemas . Ann Intern Med 1988 ; 108 : 195– 199 . 106. Von Ritter C , Grisham MB , Granger DN . Effect of 5 - aminosalicylic acid , 4 - ASA , N- acetyl - 5 - ASA and sulfapyridine on FMET - LEU - PHE induced ileitis in rats . Gastro ...
... acid enemas . Ann Intern Med 1988 ; 108 : 195– 199 . 106. Von Ritter C , Grisham MB , Granger DN . Effect of 5 - aminosalicylic acid , 4 - ASA , N- acetyl - 5 - ASA and sulfapyridine on FMET - LEU - PHE induced ileitis in rats . Gastro ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro