Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 221
... action on immune processes . Annu Rev Pharmacol Toxicol 1979 ; 19 : 179–201 . 96. Vane JR . Inhibition of prostaglandin synthesis as a mechanism of action for aspirin- like drugs . Nature New Biol 1971 ; 231 : 232-235 . 97. Lewis GP ...
... action on immune processes . Annu Rev Pharmacol Toxicol 1979 ; 19 : 179–201 . 96. Vane JR . Inhibition of prostaglandin synthesis as a mechanism of action for aspirin- like drugs . Nature New Biol 1971 ; 231 : 232-235 . 97. Lewis GP ...
Page 282
... action , however , are not completely understood . Its predom- inant action is believed to be dependent on azo bond reduction by colonic bacteria , releasing the active moiety 5 - ASA , which inhibits AA metabolism ( see Fig . 14.1 ) ...
... action , however , are not completely understood . Its predom- inant action is believed to be dependent on azo bond reduction by colonic bacteria , releasing the active moiety 5 - ASA , which inhibits AA metabolism ( see Fig . 14.1 ) ...
Page 287
... actions on calmodulin . 179 Finally , a2 adrenergic agonists in vitro are potent inhibitors of calcium - mediated secretion . Their inhibitory action may occur distal to the stimulated increases in cytosolic calcium . Another ...
... actions on calmodulin . 179 Finally , a2 adrenergic agonists in vitro are potent inhibitors of calcium - mediated secretion . Their inhibitory action may occur distal to the stimulated increases in cytosolic calcium . Another ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro