Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 510
... Acute Self - Limited Colitis Both acute self - limited colitis ( acute infectious colitis ) and idiopathic IBD pres- ent with diarrhea , which may be bloody or nonbloody . Initially , these two con- ditions can be clinically ...
... Acute Self - Limited Colitis Both acute self - limited colitis ( acute infectious colitis ) and idiopathic IBD pres- ent with diarrhea , which may be bloody or nonbloody . Initially , these two con- ditions can be clinically ...
Page 513
Richard P. MacDermott, William F. Stenson. Ischemic Colitis Clinically , acute ischemic colitis can closely mimic both active UC and acute self - limited colitis . One common situation where this diagnostic problem may be encountered is ...
Richard P. MacDermott, William F. Stenson. Ischemic Colitis Clinically , acute ischemic colitis can closely mimic both active UC and acute self - limited colitis . One common situation where this diagnostic problem may be encountered is ...
Page 601
... acute obstruction , the lesions must be differentiated from carcinoma by biopsy or excision , d particular attention should be given to ruling out Crohn's disease . Strictures ised by carcinoma are less common than those caused by ...
... acute obstruction , the lesions must be differentiated from carcinoma by biopsy or excision , d particular attention should be given to ruling out Crohn's disease . Strictures ised by carcinoma are less common than those caused by ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro