Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 209
... addition to mucus composition and secretion.65-71 In particular , PGs have been impli- cated in " intestinal cytoprotection . " This term is commonly applied to denote the mechanisms by which exogenous PGs apparently protect animals ...
... addition to mucus composition and secretion.65-71 In particular , PGs have been impli- cated in " intestinal cytoprotection . " This term is commonly applied to denote the mechanisms by which exogenous PGs apparently protect animals ...
Page 213
... addition to decreased enzymatic degradation of PGs in the presence of sulfasalazine ( but not 5 - ASA ) . 73,106-109 Nevertheless , the effect of sulfasalazine on PG formation may vary depending on substrate arachidonic acid ...
... addition to decreased enzymatic degradation of PGs in the presence of sulfasalazine ( but not 5 - ASA ) . 73,106-109 Nevertheless , the effect of sulfasalazine on PG formation may vary depending on substrate arachidonic acid ...
Page 312
... addition to mul- tiple distinct apomucin genes , diversity could also result from a genetically determined variation in the number of sequence repeats coding for important mucin functional groups . Such a variation could provide a basis ...
... addition to mul- tiple distinct apomucin genes , diversity could also result from a genetically determined variation in the number of sequence repeats coding for important mucin functional groups . Such a variation could provide a basis ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro