Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 66
... Autoimmunity The flip side of polyclonal activation , as well as a consequence of immuno- dysregulation , is the finding of autoimmune responses . Classic autoimmune dis- eases are characterized by pathogenic autoantibodies , obviously ...
... Autoimmunity The flip side of polyclonal activation , as well as a consequence of immuno- dysregulation , is the finding of autoimmune responses . Classic autoimmune dis- eases are characterized by pathogenic autoantibodies , obviously ...
Page 83
... autoimmune pathway of tissue damage . These include the lack of a convincing clinical association with other disorders with a proven autoimmune basis73 and the lack of histologic evidence for a specific cellular target for the immune ...
... autoimmune pathway of tissue damage . These include the lack of a convincing clinical association with other disorders with a proven autoimmune basis73 and the lack of histologic evidence for a specific cellular target for the immune ...
Page 406
... Autoimmune Chronic Active Hepatitis Autoimmune chronic active hepatitis occurs on average in 5 % of patients with inflammatory bowel disease ( range : 2 % to 13 % 11 ) . Autoimmune chronic active hepatitis is more common in ulcerative ...
... Autoimmune Chronic Active Hepatitis Autoimmune chronic active hepatitis occurs on average in 5 % of patients with inflammatory bowel disease ( range : 2 % to 13 % 11 ) . Autoimmune chronic active hepatitis is more common in ulcerative ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro