Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 244
... bacteria ( eg , L - forms ) increased . Two studies have demon- strated a high recovery of bacterial L - forms from gut tissue in both CD and UC15 , 16 in up to 40 % to 50 % of samples , compared with tissue from control subjects ( 1 ...
... bacteria ( eg , L - forms ) increased . Two studies have demon- strated a high recovery of bacterial L - forms from gut tissue in both CD and UC15 , 16 in up to 40 % to 50 % of samples , compared with tissue from control subjects ( 1 ...
Page 245
... bacteria . Bacterial Products Network The commensal intestinal flora secrete or contain within them a number of potent inflammatory products such as lipopolysaccharide ( LPS or endotoxin ) , peptidoglycan - polysaccharide complexes ...
... bacteria . Bacterial Products Network The commensal intestinal flora secrete or contain within them a number of potent inflammatory products such as lipopolysaccharide ( LPS or endotoxin ) , peptidoglycan - polysaccharide complexes ...
Page 257
... bacterial overgrowth . Gastroenterology 1990 ; 98 : 1-10 . 52. Vermeulen MW , Gray GR . Processing of Bacillus subtilis peptidoglycan by a mouse macrophage cell line . Infect Immun 1984 ; 46 : 476-483 . 53. Tomasic J , Ladesic B ...
... bacterial overgrowth . Gastroenterology 1990 ; 98 : 1-10 . 52. Vermeulen MW , Gray GR . Processing of Bacillus subtilis peptidoglycan by a mouse macrophage cell line . Infect Immun 1984 ; 46 : 476-483 . 53. Tomasic J , Ladesic B ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro