Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 440
... biopsies of endoscopically normal rectal mucosa , a recent study by Pötze et al . demon- strated that ulcer biopsies were more likely to demonstrate granulomas ( 29 of 85 ) than aphthoid lesions ( 7 of 32 ) or cobblestone areas ( 0 of ...
... biopsies of endoscopically normal rectal mucosa , a recent study by Pötze et al . demon- strated that ulcer biopsies were more likely to demonstrate granulomas ( 29 of 85 ) than aphthoid lesions ( 7 of 32 ) or cobblestone areas ( 0 of ...
Page 514
... biopsies and applied to clinical decision making will vary considerably from case to case and also with the stage of the disease . Generally speaking , biopsies taken at the time the patient first presents are likely to be the most ...
... biopsies and applied to clinical decision making will vary considerably from case to case and also with the stage of the disease . Generally speaking , biopsies taken at the time the patient first presents are likely to be the most ...
Page 515
... biopsies have no value . Because UC is a diagnosis of exclusion , se- quential biopsies during the course of the disease strengthen the certainty of diagnosis , which might be valuable when planning the type and extent of bowel ...
... biopsies have no value . Because UC is a diagnosis of exclusion , se- quential biopsies during the course of the disease strengthen the certainty of diagnosis , which might be valuable when planning the type and extent of bowel ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro