Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 79
... Blood Cytotoxic Mechanism Intestinal Lamina Propria Peripheral Blood Reference Natural killer CD56 + , CD16- CD56 + , CD16 + Lymphokine - activated killer CD56 , CD16- Cytotoxic T lymphocyte ( anti - CD3- triggered ) CD57 , CD5 + CD56 + ...
... Blood Cytotoxic Mechanism Intestinal Lamina Propria Peripheral Blood Reference Natural killer CD56 + , CD16- CD56 + , CD16 + Lymphokine - activated killer CD56 , CD16- Cytotoxic T lymphocyte ( anti - CD3- triggered ) CD57 , CD5 + CD56 + ...
Page 80
... blood lymphocytes , which have vigorous ADCC activity , the ability of mucosal lymphocytes to mediate ADCC has been contro- versial.23-27 Variability in results may be technical , perhaps reflecting the influ- ence of the enzymatic ...
... blood lymphocytes , which have vigorous ADCC activity , the ability of mucosal lymphocytes to mediate ADCC has been contro- versial.23-27 Variability in results may be technical , perhaps reflecting the influ- ence of the enzymatic ...
Page 160
... blood lymphocytes from patients with colorectal tumors . Immunobiology 1984 ; 166 : 494-499 . 142. Mutchnick MG , Lee HH , Hollander DI , Haynes GD , Chua DC . Defective in vitro gamma interferon production and elevated serum ...
... blood lymphocytes from patients with colorectal tumors . Immunobiology 1984 ; 166 : 494-499 . 142. Mutchnick MG , Lee HH , Hollander DI , Haynes GD , Chua DC . Defective in vitro gamma interferon production and elevated serum ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro