Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 79
... cellular immune function probably cannot be reliably extrapolated to the human . " Therefore , before examination of ... Cellular Cytotoxicity As discussed later , it has been suggested by several proponents of an autoim- mune ...
... cellular immune function probably cannot be reliably extrapolated to the human . " Therefore , before examination of ... Cellular Cytotoxicity As discussed later , it has been suggested by several proponents of an autoim- mune ...
Page 124
... cellular interactions involved in IgA biosynthesis and immune response . Adv Immunol 1987 ; 40 : 153–245 . 8. Brandtzaeg P , Korsrud FR . Significance of different J chain profiles in human tissues : Generation of IgA and IgM with ...
... cellular interactions involved in IgA biosynthesis and immune response . Adv Immunol 1987 ; 40 : 153–245 . 8. Brandtzaeg P , Korsrud FR . Significance of different J chain profiles in human tissues : Generation of IgA and IgM with ...
Page 169
... cellular turnover and vigorous remodeling of cellular and intercellular components . Inflammatory mediators of a wide variety are released in abundance . Consumption of the antigenic nidus slows these processes . Modulation In murine ...
... cellular turnover and vigorous remodeling of cellular and intercellular components . Inflammatory mediators of a wide variety are released in abundance . Consumption of the antigenic nidus slows these processes . Modulation In murine ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro