Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 325
... collagen that can be produced by smooth muscle cells , among other types . It is reasonable to hypothesize that perturbations in the abundance of , or in the relationship between , these different collagen types in the bowel might ...
... collagen that can be produced by smooth muscle cells , among other types . It is reasonable to hypothesize that perturbations in the abundance of , or in the relationship between , these different collagen types in the bowel might ...
Page 327
... collagen synthesis . The types of collagen produced by the HISM cells in culture were the same as those collagens extracted from human bowel ( types I , III , and V ) .6 These cell culture studies demonstrated that intestinal smooth ...
... collagen synthesis . The types of collagen produced by the HISM cells in culture were the same as those collagens extracted from human bowel ( types I , III , and V ) .6 These cell culture studies demonstrated that intestinal smooth ...
Page 328
... collagen production when HISM cells were exposed to these agents for 24 to 48 hours . Forskolin , an agent that also elevates cyclic AMP levels but by a different mechanism ( activation of adenylate cyclase ) , similarly inhibited collagen ...
... collagen production when HISM cells were exposed to these agents for 24 to 48 hours . Forskolin , an agent that also elevates cyclic AMP levels but by a different mechanism ( activation of adenylate cyclase ) , similarly inhibited collagen ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro