Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 180
... demonstrated a progressive movement of pigmented macrophages from superficial to deeper layers of the lamina propria ... demonstrated uptake of luminal carbon particles both by the epithelium covering Peyer's patches and by the ...
... demonstrated a progressive movement of pigmented macrophages from superficial to deeper layers of the lamina propria ... demonstrated uptake of luminal carbon particles both by the epithelium covering Peyer's patches and by the ...
Page 324
... demonstrate elastin . Control sections demonstrated a thin and distinct muscularis mucosae and a submucosa consisting of a loosely arranged network of vessels and collagen . Sections taken through strictured intestine demonstrated ...
... demonstrate elastin . Control sections demonstrated a thin and distinct muscularis mucosae and a submucosa consisting of a loosely arranged network of vessels and collagen . Sections taken through strictured intestine demonstrated ...
Page 541
... demonstrated that slices of small intestine could convert glutamine to alanine . 104 Windmueller and Spaeth 105 found that up to 33 % of the plasma glutamine was extracted in each pass through the intestine , and almost 70 % of the ...
... demonstrated that slices of small intestine could convert glutamine to alanine . 104 Windmueller and Spaeth 105 found that up to 33 % of the plasma glutamine was extracted in each pass through the intestine , and almost 70 % of the ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro