Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 342
... develop along the mesenteric border . Three days after high - dose therapy the jejunum becomes dramatically thickened and adherent to adjacent loops and frequently becomes obstructed . Adhesions develop to the serosal base of ulcers ...
... develop along the mesenteric border . Three days after high - dose therapy the jejunum becomes dramatically thickened and adherent to adjacent loops and frequently becomes obstructed . Adhesions develop to the serosal base of ulcers ...
Page 344
... develop a wasting syndrome characterized by weight loss , diarrhea , anorexia , and death . The chronic colitis is ... developed mod- erate colitis within 1 year of confinement.54 These observations have prompted speculation that the ...
... develop a wasting syndrome characterized by weight loss , diarrhea , anorexia , and death . The chronic colitis is ... developed mod- erate colitis within 1 year of confinement.54 These observations have prompted speculation that the ...
Page 587
... develop an enteric - coated preparation142 for oral delivery , no clinical studies are yet available to assess the potential in IBD . Beclometha- sone dipropionate also can be formulated with mesalamine in a combination enema for ...
... develop an enteric - coated preparation142 for oral delivery , no clinical studies are yet available to assess the potential in IBD . Beclometha- sone dipropionate also can be formulated with mesalamine in a combination enema for ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro