Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 441
... diagnosis of Crohn's disease than in chronic ulcerative colitis , where sigmoidoscopy and double - contrast barium enema establish the diagnosis in greater than 95 % of cases . 9-13 Whereas barium contrast studies may be complementary ...
... diagnosis of Crohn's disease than in chronic ulcerative colitis , where sigmoidoscopy and double - contrast barium enema establish the diagnosis in greater than 95 % of cases . 9-13 Whereas barium contrast studies may be complementary ...
Page 493
... diagnosis of inflammatory bowel disease is enhanced and facili- tated by cooperation and communication between the gastroenterologist and the pathologist . This seems so obvious , but , unfortunately , it is not always the usual ...
... diagnosis of inflammatory bowel disease is enhanced and facili- tated by cooperation and communication between the gastroenterologist and the pathologist . This seems so obvious , but , unfortunately , it is not always the usual ...
Page 515
... diagnosis of exclusion , se- quential biopsies during the course of the disease strengthen the certainty of diagnosis , which might be valuable when planning the type and extent of bowel resection should this be necessary . Morphologic ...
... diagnosis of exclusion , se- quential biopsies during the course of the disease strengthen the certainty of diagnosis , which might be valuable when planning the type and extent of bowel resection should this be necessary . Morphologic ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro