Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 98
... differentiation to J chain - positive ( J * ) plasma cells dominated by the IgA ( a * ) and IgM ( μ † ) classes . ( 10 ) SC - mediated poly - lg transport through crypt epithelium to afford immune exclusion by secretory IgA and ...
... differentiation to J chain - positive ( J * ) plasma cells dominated by the IgA ( a * ) and IgM ( μ † ) classes . ( 10 ) SC - mediated poly - lg transport through crypt epithelium to afford immune exclusion by secretory IgA and ...
Page 143
... differentiation factor ( EDF ) , 78 and promotes eosinophil precursor growth and differentiation , an activity amplified by the concomitant input of IL - 1 and IL - 3.79 Very little is known about IL - 5 in humans . Interleukin - 6 The ...
... differentiation factor ( EDF ) , 78 and promotes eosinophil precursor growth and differentiation , an activity amplified by the concomitant input of IL - 1 and IL - 3.79 Very little is known about IL - 5 in humans . Interleukin - 6 The ...
Page 145
... differentiation and cell growth inhibition , enhancement of nat- ural killer and antibody - dependent cytotoxicity , macrophage activation , mod- ulation of B - cell responses , inhibition of suppressor cells , and enhancement of T cell ...
... differentiation and cell growth inhibition , enhancement of nat- ural killer and antibody - dependent cytotoxicity , macrophage activation , mod- ulation of B - cell responses , inhibition of suppressor cells , and enhancement of T cell ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro