Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 62
... cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access to a larger number of luminal antigens may result . The second ...
... cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access to a larger number of luminal antigens may result . The second ...
Page 85
... cell hyperplasia may be stimulated by cy- tokines from activated T cells and macrophages . Organ culture studies have shown that changes in crypt epithelial proliferation resulting from the activation of lamina propria T cells by anti ...
... cell hyperplasia may be stimulated by cy- tokines from activated T cells and macrophages . Organ culture studies have shown that changes in crypt epithelial proliferation resulting from the activation of lamina propria T cells by anti ...
Page 91
... cells for autologous colonic epithelial cells in colonic diseases . Gastroenterology 1984 ; 86 : 13–22 . 66. Gibson PR , van de Pol E , Pullman W , Doe WF . Lysis of colonic epithelial cells by allogeneic mononuclear and lymphokine ...
... cells for autologous colonic epithelial cells in colonic diseases . Gastroenterology 1984 ; 86 : 13–22 . 66. Gibson PR , van de Pol E , Pullman W , Doe WF . Lysis of colonic epithelial cells by allogeneic mononuclear and lymphokine ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro