Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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... et al . Macrocortin : A polypeptide causing the antiphospholipase effect of glucocorticoids . Nature 1980 ; 287 : 147–149 . 133. DiRosa M , Flower RJ , Hirata F , et al . Nomenclature announcement : Anti - phospho- lipase proteins ...
... et al . Macrocortin : A polypeptide causing the antiphospholipase effect of glucocorticoids . Nature 1980 ; 287 : 147–149 . 133. DiRosa M , Flower RJ , Hirata F , et al . Nomenclature announcement : Anti - phospho- lipase proteins ...
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... et al 197327 7 NR 3 ( 43 % ) NR NR Additional medical therapy Vogel et al 197428 8 9-50 8 ( 100 % ) 4 ( 50 % ) 4-36 Additional medical therapy Eisenberg et al./197429 9 Fazio et al./197630 and 18 120 21 5 ( 55 % ) NR NR 20 11 ( 61 % ) 4 ...
... et al 197327 7 NR 3 ( 43 % ) NR NR Additional medical therapy Vogel et al 197428 8 9-50 8 ( 100 % ) 4 ( 50 % ) 4-36 Additional medical therapy Eisenberg et al./197429 9 Fazio et al./197630 and 18 120 21 5 ( 55 % ) NR NR 20 11 ( 61 % ) 4 ...
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... et al 197356 Rocchio et al./197457 Segal et al 197758 Axelson and Jarnum / 197759 Prospective , uncontrolled studies O'Morain et al./198060 Logan et al 198161 Prospective , randomized , Number of Patients Duration of EN ( days ) ...
... et al 197356 Rocchio et al./197457 Segal et al 197758 Axelson and Jarnum / 197759 Prospective , uncontrolled studies O'Morain et al./198060 Logan et al 198161 Prospective , randomized , Number of Patients Duration of EN ( days ) ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro