Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 25
... evidence of anky- losing spondylitis.103 Among the first - degree relatives of 142 Liverpool IBD pa- tients , there were 3 with ankylosing spondylitis , none of whom had evidence of IBD clinically or on barium enema examination and ...
... evidence of anky- losing spondylitis.103 Among the first - degree relatives of 142 Liverpool IBD pa- tients , there were 3 with ankylosing spondylitis , none of whom had evidence of IBD clinically or on barium enema examination and ...
Page 73
... Evidence of virus infection as a cause of Crohn's disease . Z Gastroenterol Suppl 1979 ; 17 : 101 . 112. Sewell P , Cooke WT , Cox EV , Meynell MJ . Milk tolerance in gastrointestinal disorders . Lancet 1963 ; 2 : 1132 . 113. Dudek B ...
... Evidence of virus infection as a cause of Crohn's disease . Z Gastroenterol Suppl 1979 ; 17 : 101 . 112. Sewell P , Cooke WT , Cox EV , Meynell MJ . Milk tolerance in gastrointestinal disorders . Lancet 1963 ; 2 : 1132 . 113. Dudek B ...
Page 245
... evidence exists for a role of the commensal flora in nonsteroidal anti - inflammatory drug ( NSAID ) -induced ileitis , 25 radiation - in- duced colitis , graft - versus - host disease of the intestine , and ischemic colitis in ...
... evidence exists for a role of the commensal flora in nonsteroidal anti - inflammatory drug ( NSAID ) -induced ileitis , 25 radiation - in- duced colitis , graft - versus - host disease of the intestine , and ischemic colitis in ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro