Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 83
... experiments suffered from several technical limitations . The effector cell was not well characterized and the evidence for epithelial lysis was based on microscopic examination of crude isolates of in- testinal epithelial cells , which ...
... experiments suffered from several technical limitations . The effector cell was not well characterized and the evidence for epithelial lysis was based on microscopic examination of crude isolates of in- testinal epithelial cells , which ...
Page 265
... experiment is at first sight impressive and the negative results would seem to exclude the possibility of mycobacterial infection in a significant number of CD patients ; however , when similar experiments were performed on DNA ...
... experiment is at first sight impressive and the negative results would seem to exclude the possibility of mycobacterial infection in a significant number of CD patients ; however , when similar experiments were performed on DNA ...
Page 349
... experimental injury may have some relevance to inflammatory bowel disease ; however , failure of the standard IBD drugs , cor- ticosteroids and sulfasalazine , to diminish inflammation in many of the models of experimental intestinal ...
... experimental injury may have some relevance to inflammatory bowel disease ; however , failure of the standard IBD drugs , cor- ticosteroids and sulfasalazine , to diminish inflammation in many of the models of experimental intestinal ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro