Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 119
... Expression HLA - DR is normally expressed by the small intestinal villous epithelium but generally not by colonic crypt or surface epithelium.226 In various gut disorders epithelial class II expression is often markedly enhanced . Selby ...
... Expression HLA - DR is normally expressed by the small intestinal villous epithelium but generally not by colonic crypt or surface epithelium.226 In various gut disorders epithelial class II expression is often markedly enhanced . Selby ...
Page 135
... expression and disease activity in ulcerative colitis . Scand J Gastroenterol 1986 ; 21 : 364–368 . 229. McDonald GB , Jewell DP . Class II antigen ( HLA - DR ) expression by intestinal epi- thelial cells in inflammatory diseases of ...
... expression and disease activity in ulcerative colitis . Scand J Gastroenterol 1986 ; 21 : 364–368 . 229. McDonald GB , Jewell DP . Class II antigen ( HLA - DR ) expression by intestinal epi- thelial cells in inflammatory diseases of ...
Page 179
... expression of the class II major histocompatibility complex ( MHC ) antigens ( in the order DR , DP , and DQ ) , the receptor for interleukin - 2 ( IL - 2 ) , and decreased expression of the surface receptors for the Fc component of IgG ...
... expression of the class II major histocompatibility complex ( MHC ) antigens ( in the order DR , DP , and DQ ) , the receptor for interleukin - 2 ( IL - 2 ) , and decreased expression of the surface receptors for the Fc component of IgG ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro