Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 109
... ileitis ( n = 4 ) IgA 79 % ( x2.8 ) IgM 13 % ( x3.3 ) 134 IC / unit IgA 53 % ( x1.7 ) ( x3 ) IgG 8 % ( x4.1 ) Normal large bowel ( n = 19 ) 143 IC / unit IgM 4 % ( x2.1 ) IgG 43 % ( x31 ) Active ( severe to moderate ) ulcerative colitis ...
... ileitis ( n = 4 ) IgA 79 % ( x2.8 ) IgM 13 % ( x3.3 ) 134 IC / unit IgA 53 % ( x1.7 ) ( x3 ) IgG 8 % ( x4.1 ) Normal large bowel ( n = 19 ) 143 IC / unit IgM 4 % ( x2.1 ) IgG 43 % ( x31 ) Active ( severe to moderate ) ulcerative colitis ...
Page 264
... ileitis , " and all were found to be identical to CD - derived M. paratuberculosis . 14 We have examined more than 100 M. avium group strains isolated ( many using myco- bactin included in media ) from Western AIDS patients and from the ...
... ileitis , " and all were found to be identical to CD - derived M. paratuberculosis . 14 We have examined more than 100 M. avium group strains isolated ( many using myco- bactin included in media ) from Western AIDS patients and from the ...
Page 510
... Ileitis Backwash ileitis can occur in up to 20 % of patients who have UC involving the whole colon . Histologically , the character and distribution of the inflammation are exactly the same as for colonic involvement ; that is , it is ...
... Ileitis Backwash ileitis can occur in up to 20 % of patients who have UC involving the whole colon . Histologically , the character and distribution of the inflammation are exactly the same as for colonic involvement ; that is , it is ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro