Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 53
... immune regulation . Although the immune system had been indirectly impli- cated since the late 1960s , 1,2 more recent findings from several groups speak for an active poorly controlled immune response occurring in the gastrointes ...
... immune regulation . Although the immune system had been indirectly impli- cated since the late 1960s , 1,2 more recent findings from several groups speak for an active poorly controlled immune response occurring in the gastrointes ...
Page 65
... immune complexes adequately . In addition , cor- relation of immune complexes in the serum may not reflect what is actually going on in the tissue . With use of conventional assays , such as the RAJI cell assay and C1q precipitation ...
... immune complexes adequately . In addition , cor- relation of immune complexes in the serum may not reflect what is actually going on in the tissue . With use of conventional assays , such as the RAJI cell assay and C1q precipitation ...
Page 124
... immune mechanisms in the pathogenesis of Crohn's disease . In Vivo 1988 ; 2 : 1-8 . 4. Brandtzaeg P , Halstensen TS , Kett K , et al . Immunobiology and immunopathology of human gut mucosa : Humoral immunity and intraepithelial ...
... immune mechanisms in the pathogenesis of Crohn's disease . In Vivo 1988 ; 2 : 1-8 . 4. Brandtzaeg P , Halstensen TS , Kett K , et al . Immunobiology and immunopathology of human gut mucosa : Humoral immunity and intraepithelial ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro