Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 53
Richard P. MacDermott, William F. Stenson. Chapter 3 Mucosal Immune System in Inflammatory Bowel Disease Lloyd Mayer , MD Over the past decade our understanding of the gut ... Immune System in Inflammatory Bowel Disease / Lloyd Mayer, MD.
Richard P. MacDermott, William F. Stenson. Chapter 3 Mucosal Immune System in Inflammatory Bowel Disease Lloyd Mayer , MD Over the past decade our understanding of the gut ... Immune System in Inflammatory Bowel Disease / Lloyd Mayer, MD.
Page 124
... immune response . Adv Immunol 1987 ; 40 : 153–245 . 8. Brandtzaeg P , Korsrud FR . Significance of different J chain profiles in human tissues : Generation of IgA and IgM with binding site for secretory component is related to the J ...
... immune response . Adv Immunol 1987 ; 40 : 153–245 . 8. Brandtzaeg P , Korsrud FR . Significance of different J chain profiles in human tissues : Generation of IgA and IgM with binding site for secretory component is related to the J ...
Page 241
... response depend on recognition of products of the metabolism of such microorganisms . The more obvious defects of the immune system are now well characterized and can be congenital or acquired . As far as interactions with ...
... response depend on recognition of products of the metabolism of such microorganisms . The more obvious defects of the immune system are now well characterized and can be congenital or acquired . As far as interactions with ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro