Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 21
... important clues regarding the contributions of genetic susceptibility and environmental exposure . If genetic predisposition is not important in the etiology of a disease , then spouses , who theoretically should be exposed to similar ...
... important clues regarding the contributions of genetic susceptibility and environmental exposure . If genetic predisposition is not important in the etiology of a disease , then spouses , who theoretically should be exposed to similar ...
Page 315
... important defense prop- erties . These include a close association with IgA , which may confer antimi- crobial and antitoxic properties to the mucus gel overlying the mucosa.49 This may be especially important in the neonatal period ...
... important defense prop- erties . These include a close association with IgA , which may confer antimi- crobial and antitoxic properties to the mucus gel overlying the mucosa.49 This may be especially important in the neonatal period ...
Page 528
... important to differentiate between a normally thin individual with a normal energy level and a nutritionally depleted one with muscular weakness . Malnutrition that impairs body function significantly im- pairs clinical outcome . 5 ...
... important to differentiate between a normally thin individual with a normal energy level and a nutritionally depleted one with muscular weakness . Malnutrition that impairs body function significantly im- pairs clinical outcome . 5 ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro