Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 151
... Increased Increased IL - 2 Undetectable Decreased IFN - Y Undetectable Decreased CSF Increased Increased IL - 4 ? ? IL - 5 ? ? IL - 6 Increased ? IL - 7 IL - 8 TNF - α TNF - B PAF TGF - a ? Increased Comparable / undetectable ( ? ) ?
... Increased Increased IL - 2 Undetectable Decreased IFN - Y Undetectable Decreased CSF Increased Increased IL - 4 ? ? IL - 5 ? ? IL - 6 Increased ? IL - 7 IL - 8 TNF - α TNF - B PAF TGF - a ? Increased Comparable / undetectable ( ? ) ?
Page 206
... increased activity of PG synthetase . The amounts of PGE2 , PGF2a , 6 - keto - PGF1a ( the stable breakdown product of PGI2 ) , TXB2 ( the stable breakdown product of TXA2 ) , 12 - HETE , 15 - HETE , LTB4 , and LTC4 generated by ...
... increased activity of PG synthetase . The amounts of PGE2 , PGF2a , 6 - keto - PGF1a ( the stable breakdown product of PGI2 ) , TXB2 ( the stable breakdown product of TXA2 ) , 12 - HETE , 15 - HETE , LTB4 , and LTC4 generated by ...
Page 279
... increased permeability to these markers . " Furthermore , increased separation of tight junctions and abnormalities in junctional strands have been reported in the noninflamed intestine . 73,74 In the inflamed intestine , it is likely ...
... increased permeability to these markers . " Furthermore , increased separation of tight junctions and abnormalities in junctional strands have been reported in the noninflamed intestine . 73,74 In the inflamed intestine , it is likely ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro