Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 225
... Injury Matthew B. Grisham , PhD and D. Neil Granger , PhD Active episodes of inflammation that occur in inflammatory bowel disease ( Crohn's disease and ulcerative colitis ) are characterized by the extravasation and infiltration of ...
... Injury Matthew B. Grisham , PhD and D. Neil Granger , PhD Active episodes of inflammation that occur in inflammatory bowel disease ( Crohn's disease and ulcerative colitis ) are characterized by the extravasation and infiltration of ...
Page 330
... injury from the mucosal surface . Mucosal injury is repaired by a process of rapid restitution of the epithelial lining.22 This is accomplished without a mesenchymal / connective tissue re- sponse and therefore without any morphologic ...
... injury from the mucosal surface . Mucosal injury is repaired by a process of rapid restitution of the epithelial lining.22 This is accomplished without a mesenchymal / connective tissue re- sponse and therefore without any morphologic ...
Page 343
... injury . In the stomach , acid , pepsin , and perhaps bile acids produce injury ; normal bacterial flora and bacterial products such as peptidoglycan polymers appear to be im- portant in the small intestine ; in the cecum viable ...
... injury . In the stomach , acid , pepsin , and perhaps bile acids produce injury ; normal bacterial flora and bacterial products such as peptidoglycan polymers appear to be im- portant in the small intestine ; in the cecum viable ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro