Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 261
... involved . Genetic analysis of rare families in which , because of " nor- malization " of other factors across the family , CD may behave as a single Men- delian trait may allow us to identify genes involved in susceptibility . As well ...
... involved . Genetic analysis of rare families in which , because of " nor- malization " of other factors across the family , CD may behave as a single Men- delian trait may allow us to identify genes involved in susceptibility . As well ...
Page 269
... involved in the pathology . Second , the drug regimens used may not be effective against the mycobacteria involved . If atypical mycobacteria , such as M. avium , are involved in CD , then experience with atypical mycobacterial ...
... involved in the pathology . Second , the drug regimens used may not be effective against the mycobacteria involved . If atypical mycobacteria , such as M. avium , are involved in CD , then experience with atypical mycobacterial ...
Page 621
... involved area , thickened mesentery prevents palpation of mesenteric edge of intestine Extent of surgical resection determined by palpation of mesentery . Only significantly involved segment excised to prevent creation of short - bowel ...
... involved area , thickened mesentery prevents palpation of mesenteric edge of intestine Extent of surgical resection determined by palpation of mesentery . Only significantly involved segment excised to prevent creation of short - bowel ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro