Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 298
... jejunum . J Pharmacol Exp Ther 1982 ; 220 : 637–641 . 188. Chang EB , Gill AJ , Wang NS , Field M. Alpha - 2 - adrenergic inhibition of Ca - dependent intestinal secretion . Gastroenterology 1984 ; 86 : A1044 . Abstract . 189. McArthur ...
... jejunum . J Pharmacol Exp Ther 1982 ; 220 : 637–641 . 188. Chang EB , Gill AJ , Wang NS , Field M. Alpha - 2 - adrenergic inhibition of Ca - dependent intestinal secretion . Gastroenterology 1984 ; 86 : A1044 . Abstract . 189. McArthur ...
Page 342
... jejunum becomes dramatically thickened and adherent to adjacent loops and frequently becomes obstructed . Adhesions develop to the serosal base of ulcers , but relatively normal mucosa ( skip lesions ) is found between ulcerations ...
... jejunum becomes dramatically thickened and adherent to adjacent loops and frequently becomes obstructed . Adhesions develop to the serosal base of ulcers , but relatively normal mucosa ( skip lesions ) is found between ulcerations ...
Page 543
... jejunum and ileum . The crypt cell production rate was also sig- nificantly greater in the pectin - fed group compared with the non - pectin - fed group . Ileal mucosa - specific activities of maltase , sucrase , and alkaline phos ...
... jejunum and ileum . The crypt cell production rate was also sig- nificantly greater in the pectin - fed group compared with the non - pectin - fed group . Ileal mucosa - specific activities of maltase , sucrase , and alkaline phos ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro