Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 392
... lesions . The ulcers can become so large that they destroy surrounding tissues and can even eventually cause loss of a limb . There seems to be a relationship to trauma and needle injections prior to the onset of the cutaneous lesions ...
... lesions . The ulcers can become so large that they destroy surrounding tissues and can even eventually cause loss of a limb . There seems to be a relationship to trauma and needle injections prior to the onset of the cutaneous lesions ...
Page 393
... lesions are painful . Because activity of the oral lesions parallels the intestinal disease activity there is frequently a remission of the oral lesions following successful therapy for the underlying intestinal disease . Another oral ...
... lesions are painful . Because activity of the oral lesions parallels the intestinal disease activity there is frequently a remission of the oral lesions following successful therapy for the underlying intestinal disease . Another oral ...
Page 446
... lesions in 13 children ( 42 % ) and these were confirmed histologically in 39 % ( granulomas 39 % , non- specific inflammation 48 % ) . Aphthoid ulcerations were most common but pol- ypoid and inflammatory gastroduodenitis were also ...
... lesions in 13 children ( 42 % ) and these were confirmed histologically in 39 % ( granulomas 39 % , non- specific inflammation 48 % ) . Aphthoid ulcerations were most common but pol- ypoid and inflammatory gastroduodenitis were also ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro