Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 179
... macrophage function can be inferred . Intestinal Macrophages Intestinal macrophages constitute 10 % to 20 % of the total cell number in the lamina propria as determined by immunocytochemistry and tissue disaggre- gation experiments.5.6 ...
... macrophage function can be inferred . Intestinal Macrophages Intestinal macrophages constitute 10 % to 20 % of the total cell number in the lamina propria as determined by immunocytochemistry and tissue disaggre- gation experiments.5.6 ...
Page 180
... macrophages migrated through fenestrations in the epithelial cell basement membrane to the superficial lamina propria . Analysis of the time course of macrophage migration over a 2 - month period following the cessation of danthron ...
... macrophages migrated through fenestrations in the epithelial cell basement membrane to the superficial lamina propria . Analysis of the time course of macrophage migration over a 2 - month period following the cessation of danthron ...
Page 183
Richard P. MacDermott, William F. Stenson. macrophage involves TGF - ẞ , which is also a product of activated macrophages and may provide , along with PGE2 , a negative feedback loop.4 Oxyradicals 45 Of the oxyradicals , including ...
Richard P. MacDermott, William F. Stenson. macrophage involves TGF - ẞ , which is also a product of activated macrophages and may provide , along with PGE2 , a negative feedback loop.4 Oxyradicals 45 Of the oxyradicals , including ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro