Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 27
... Marker Studies The early genetic marker studies using polymorphic blood group markers found no evidence for either linkage or association with a variety of general poly- morphic markers such as ABO , rhesus blood group , a1 ...
... Marker Studies The early genetic marker studies using polymorphic blood group markers found no evidence for either linkage or association with a variety of general poly- morphic markers such as ABO , rhesus blood group , a1 ...
Page 32
... markers on immunoglobulin G ( Gm markers ) , immu- noglobulin A ( Am markers ) , and Kappa light chains ( Km markers ) in a series of CD and UC patients and healthy controls . Although no differences were found between the UC patients ...
... markers on immunoglobulin G ( Gm markers ) , immu- noglobulin A ( Am markers ) , and Kappa light chains ( Km markers ) in a series of CD and UC patients and healthy controls . Although no differences were found between the UC patients ...
Page 33
... markers that are closer to the basic defect and thus likely to detect more individuals with the abnormal genotype . Subclinical markers are parameters used to detect the abnormal geno- type in the absence of the full phenotype , for ...
... markers that are closer to the basic defect and thus likely to detect more individuals with the abnormal genotype . Subclinical markers are parameters used to detect the abnormal geno- type in the absence of the full phenotype , for ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro