Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 79
... mechanisms that may have particular relevance to IBD is pre- sented . The cytotoxic potential of mucosal lymphocytes and mucosal T - cell function are the focus here because they have been investigated extensively as potential mediators ...
... mechanisms that may have particular relevance to IBD is pre- sented . The cytotoxic potential of mucosal lymphocytes and mucosal T - cell function are the focus here because they have been investigated extensively as potential mediators ...
Page 274
... mechanisms must be considered . In general , at least five potential mechanisms appear to play a role in the development of diarrhea : ( 1 ) stimulation of active secretion of water and electrolytes , ( 2 ) mal- absorption of solutes ...
... mechanisms must be considered . In general , at least five potential mechanisms appear to play a role in the development of diarrhea : ( 1 ) stimulation of active secretion of water and electrolytes , ( 2 ) mal- absorption of solutes ...
Page 654
... mechanisms , 228-229 oxidative mechanisms , 225-227 proposed interaction among neutrophil- derived oxidants , proteases , and protease inhibitors , 229f Neutrophils , 277 and mucosal permeability , 234-236 role as mediators of ...
... mechanisms , 228-229 oxidative mechanisms , 225-227 proposed interaction among neutrophil- derived oxidants , proteases , and protease inhibitors , 229f Neutrophils , 277 and mucosal permeability , 234-236 role as mediators of ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro