Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 79
... mucosal effector cells such as mast cells also differ functionally and phenotypically from those at nonmucosal sites ( reviewed in Chapter 9 ) . An additional feature that distinguishes the mucosal from the sys- temic cellular immune ...
... mucosal effector cells such as mast cells also differ functionally and phenotypically from those at nonmucosal sites ( reviewed in Chapter 9 ) . An additional feature that distinguishes the mucosal from the sys- temic cellular immune ...
Page 81
... mucosa and blood of patients with IBD41 ; however , the cytotoxicity induced by mitogens in culture is difficult to distinguish from culture - activated or lymphokine - activated cyto- toxicity . 15.42 Direct study of mucosal T - cell ...
... mucosa and blood of patients with IBD41 ; however , the cytotoxicity induced by mitogens in culture is difficult to distinguish from culture - activated or lymphokine - activated cyto- toxicity . 15.42 Direct study of mucosal T - cell ...
Page 653
... Mucosa 318 damage , sustained , 185-186 immune system . See Mucosal immune system indomethacin - induced injury , 343 permeability and leaky bowel , 279 significance of altered immunologic homeostasis in the , 120-122 Mucosa ...
... Mucosa 318 damage , sustained , 185-186 immune system . See Mucosal immune system indomethacin - induced injury , 343 permeability and leaky bowel , 279 significance of altered immunologic homeostasis in the , 120-122 Mucosa ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro