Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 242
... normal rodents has been estimated to be 10-3 to 10-4 of an intragastric load for intact bacterial macromolecules3 ... Normal Load or Exposure Defective Normal Aggressive Normal Aggressive Increased " Normal Increased Normal ...
... normal rodents has been estimated to be 10-3 to 10-4 of an intragastric load for intact bacterial macromolecules3 ... Normal Load or Exposure Defective Normal Aggressive Normal Aggressive Increased " Normal Increased Normal ...
Page 318
... normal mucin species IV argues against a major structural apomucin abnormality ; however , it is possible that a mutation in this mucin resides in the portion that interacts with the molecules responsible for mucin packaging and ...
... normal mucin species IV argues against a major structural apomucin abnormality ; however , it is possible that a mutation in this mucin resides in the portion that interacts with the molecules responsible for mucin packaging and ...
Page 494
... normal mucosa is interposed between inflamed segments of bowel ; however , there may be segmental variation in the intensity of inflam- mation , giving rise to an erroneous endoscopic impression that certain areas of the bowel are normal ...
... normal mucosa is interposed between inflamed segments of bowel ; however , there may be segmental variation in the intensity of inflam- mation , giving rise to an erroneous endoscopic impression that certain areas of the bowel are normal ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro